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HAMER, Pharm D, BCPP, Oregon State University College of Pharmacy, Corvallis, Oregon Am Fam Physician. Related Editorial The use of antipsychotic medications entails a difficult trade-off between the benefit of alleviating psychotic symptoms and the risk of troubling, sometimes life-shortening adverse effects.

They are more likely to occur with higher dosages of high-potency FGAs, such as haloperidol (formerly Haldol), and are less likely with FGAs that have weaker dopamine blockade.These include fluphenazine, haloperidol (formerly Haldol), thiothixene (Navane), neuroreceptor blockade, with an adverse and trifluoperazine.Please note that the FGA perphenazine is considered to have intermediate dopamine D neuroreceptor blockade.For information about the SORT evidence rating system, go to medications effectively diminish the intensity of psychotic hallucinations, allowing most institutionalized patients with schizophrenia to be discharged into community treatment.The first-generation antipsychotics (FGAs) work through dopamine D neuroreceptor blockade, and a subsequent series of new antipsychotics were developed with stronger dopamine blockade.1 To discuss differences in the adverse effect profiles of FGAs, we use the terms “low-potency” and “high-potency,” not to indicate their clinical effectiveness, but rather to indicate their potency in binding to this dopamine DSecond-generation antipsychotics (SGAs) were launched in 1989 when investigators found that clozapine (Clozaril) was more effective than chlorpromazine, with fewer extrapyramidal symptoms.2 These new anti-psychotics were considered atypical because they targeted neuroreceptors other than only dopamine.

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